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Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction.
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Fog2 is critical for cardiac function and maintenance of coronary vasculature in the adult mouse heart.
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A positive feedback loop of phosphodiesterase 3 (PDE3) and inducible cAMP early repressor (ICER) leads to cardiomyocyte apoptosis.
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